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Worse still, the prolonged hyperglycemia further causes metabolic disorders, including the aberration of the polyol pathway and hexosamine biosynthesis pathway, advanced glycation end (AGE) formation, and the activation of protein kinase C, all of which further augments ROS generation and form a vicious cycle [125, 126]

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Particular Cytoprotective Aspects of BPC 157 Therapy in Glaucoma This review attempts to reveal these particular aspects of BPC 157 therapy that may also be useful in the therapy of glaucoma