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In summary, RA is a multifaceted disease in which autoimmune, inflammatory, and oxidative processes are deeply interconnected, leading to sustained tissue damage

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Additionally, various mechanisms have been investigated to further explain the pathogenesis of AD, including cholinergic dysfunction, oxidative damage, deposits of amyloid beta (Aβ), hyperphosphorylation of tau protein and the aggregation of senile plaques (4,5)