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Nonetheless, they suggested that, in the case of renal disease, GBCAs decreased renal function [75], triggered skin fibrosis [11,57], increased the number of fibrocytes (related to the oxidative stress environment) [76], enhanced the differentiation of mononuclear cells into ferroportin-expressing fibrocystic cells [67], produced renal tube vacuolization [11], and caused disturbances in iron metabolism and TBARS values [75], as well as increased neutrophil count and neutrophil elastase activity [35]

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Specifically for PD, several studies using engineered MSCs that expressed tyrosine hydroxylase 155 , vascular endothelial growth factor 156 , or were transduced to produce increased glial cell-derived neurotrophic factor 157 or cerebral dopamine neurotrophic factor 158 have demonstrated positive results in preclinical rodent models