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Further supporting this, Lock and colleagues demonstrated that glutathione protects against 2-chloropropionic acid (CPA)-induced cerebellar toxicity by preserving thiol redox equilibrium and potentially interacting with NMDA receptor-mediated pathways
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[DOI] [PubMed] [Google Scholar] 42.Rabenstein D.L., Isab A.A., Kadima W., Mohanakrishnan P
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