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Neuroinflammation and energy metabolism: a dual perspective on ischemic stroke | Journal of Translational Medicine | Springer Nature Link
Early studies pointed to the plasma membrane as the primary execution site, supported by findings that FSP1, as described above, exerts its antiferroptotic function through the reduction of plasma membrane ubiquinone (CoQ 10 ) 16,17
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H 2 S production in mammals was known biochemically by the early 1990s
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