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However, further studies are necessary to understand the physiopathological mechanisms implicated (Schetters et al., 2018) because it is unknown whether OS is a primary cause that induces the initiation of neurodegenerative diseases or if it is a side effect associated with the spread of damage to nerve cells (Jenner, 2003

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(2023) demonstrated that L-carnitine, at concentrations of 1–3 mmol/L, effectively controls DED by inhibiting the activation of the transient receptor potential vanilloid subtype 1 (TRPV1) induced by hyperosmolarity