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Microglial exosomes alleviate intermittent hypoxia-induced cognitive deficits by suppressing NLRP3 inflammasome
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Highly significant ( P ≤ 0.001) decrease of SAM and SAH was also observed in subjects with ASD.[114] These observations were supported by detailed analysis of metabolic and nutritional status of 55 children with ASD ages 5–16 years compared with nonsibling, neurotypical controls ( n = 44).[3] Fluoride can contribute to disturbances in GSH homeostasis because it enters the cascade and attaches to the SAH
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