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TLR4 recognizes exogenous and endogenous ligands, including oxLDL, heparan sulfate, HSPs, fibrinogen, hyaluronan fragments, beta-defensin, lipoteichoic acid (LTA), lipopolysaccharide (LPS), protein F, and envelope glycoprotein, that are released at chronic inflammation sites in response to tissue stress or damage, such as atherosclerosis (Fig
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