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Decreased GSH content in the apical fluid in cystic fibrosis could be the result of abnormal GSH transport associated with a defective cystic fibrosis transmembrane conductance regulator as mentioned previously (126)
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Similarly, unmyelinated segments of retinal ganglion cell axons in the lamina cribrosa also show increased metabolic activity in comparison to myelinated segments (Bristow et al., 2002
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