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Our in vitro studies on primary peritoneal macrophages from Gstp −/− mice and wild-type mice, and cell lines of RAW264.7 and THP-1 indicated that GSTP inhibited LPS-induced HMGB1 release through preventing its translocation from the nucleus to cytoplasm
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CMA is also found to decrease CD36 S-palmitoylation, which contributes to reduce FA uptake and disturb lipid metabolism [189]
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